ABSTRACT
Acquired ileal atresia is a rare but life-threatening complication that can occur after recovery from necrotizing enterocolitis in premature infants. We present an unusual case of acquired ileal atresia in the distal ileum injured by ischemia after intestinal perforation in an extremely low-birth-weight infant. A 900 g (25 weeks gestation) premature infant developed a pneumoperitoneum without the radiologic manifestations of necrotizing enterocolitis on day 9 of life. Primary peritoneal drainage without further need for surgery was performed in the neonatal intensive care facility. Gastrointestinal gastrografin studies confirmed normal intestinal continuity and regular stools. Several weeks later, while the patient was receiving all nutrition orally with no medical problems, the patient's condition suddenly deteriorated, along with clinical signs of intestinal obstruction including emesis of bilious contents and stools. Laparotomy (on day 45 of life) revealed ileal atresia with V-shaped gap mesenteric defect.
Subject(s)
Humans , Infant, Newborn , Diatrizoate Meglumine , Drainage , Enterocolitis, Necrotizing , Ileum , Infant, Low Birth Weight , Infant, Premature , Intensive Care, Neonatal , Intestinal Obstruction , Intestinal Perforation , Ischemia , Laparotomy , Pneumoperitoneum , VomitingABSTRACT
Acquired ileal atresia is a rare but life-threatening complication that can occur after recovery from necrotizing enterocolitis in premature infants. We present an unusual case of acquired ileal atresia in the distal ileum injured by ischemia after intestinal perforation in an extremely low-birth-weight infant. A 900 g (25 weeks gestation) premature infant developed a pneumoperitoneum without the radiologic manifestations of necrotizing enterocolitis on day 9 of life. Primary peritoneal drainage without further need for surgery was performed in the neonatal intensive care facility. Gastrointestinal gastrografin studies confirmed normal intestinal continuity and regular stools. Several weeks later, while the patient was receiving all nutrition orally with no medical problems, the patient's condition suddenly deteriorated, along with clinical signs of intestinal obstruction including emesis of bilious contents and stools. Laparotomy (on day 45 of life) revealed ileal atresia with V-shaped gap mesenteric defect.